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Pharmacological Properties Escitalopram — antidepressant group of selective serotonin reuptake inhibitors, S www.studiodentisticocesanoboscone.it serotonin reverse, increasing its concentration in the synaptic cleft, resulting in increased and prolonged serotoninergic activity within the central nervous system. In studies in vivo on animals and in vitro demonstrated the ability of escitalopram highly selectively inhibit reverse neuronal uptake of serotonin in the central nervous system, the re-uptake of dopamine and noradrenaline escitalopram has minimal effect.
Escitalopram at least 100 times more potent to inhibit serotonin reuptake than the R — enantiomer. In the simulation, the antidepressant effect of tolerance does not develop during prolonged up to 5 weeks to rats. Also escitalopram it has a very low affinity or not bind to various ion channels, Non Prescription Escitalopram, including sodium, potassium, calcium, Non prescription Escitalopram channels.
The antidepressant effect of escitalopram usually develops after 2 — 4 weeks after initiation of therapy. Maximum therapeutic effect treatment of panic disorders is achieved after about 3 months after starting treatment When receiving single and multiple doses. Range 10 — 30 mg per day has a dose-dependent pharmacokinetics escitalopram and linear. Oral solution and Non prescriptions Escitalopram that contain escitalopram oxalate are bioequivalent. Absorption of escitalopram is independent of food intake. After a single oral 20 mg, the maximum concentration is reached after about 5 hours. In young healthy individuals escitalopram plasma concentration at steady state of 2. Level S — didemetiltsitaloprama in most subjects not determined. In in vitro studies have shown that inhibition of the serotonin reuptake escitalopram above that S — demetiltsitaloprama at least 7-fold, and S — didemetiltsitaloprama — 27 Non prescriptions Escitalopram.
This indicates that the metabolites do not contribute significantly to the antidepressant action of escitalopram. S — didemetiltsitaloprama and S — demetiltsitaloprama also have a very poor ability to communicate or Non prescription Escitalopram with Non prescription Escitalopram 5 — HT1-7, histamine H1-3, dopamine D1-5, benzodiazepine, muscarinic M1-5 receptors, alpha — and beta — adrenergic receptors, it does not bind to various ion channels, including sodium, potassium, calcium, chloride channels, Non Prescription Escitalopram.
In persons with a weak activity level escitalopram CYP2C19 isozyme may be two times higher than those with a high activity of this isoenzyme. The terminal half-life of approximately 27 — 32 hours. In the Non prescription Escitalopram metabolites of escitalopram half-life is longer. Time of maximum concentration, half-life in depending on the floor. For most patients with impaired hepatic function The recommended dose is 10 mg escitalopram. Indications The depression, major depressive disorder, panic disorder including with agoraphobia, generalized anxiety disorder.
How to use and dosages of escitalopram Escitalopram is taken by mouth, with or without food once a day. The course of therapy lasting, established by the attending physician. Depression and certain other psychiatric disorders are associated with an increased risk of suicide. When receiving antidepressants cheap Mestinon antidepressant in Non prescriptions Escitalopram age groups, Non Prescription Escitalopram.
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At joint use of medicines from the group of SSRIs together with monoamine oxidase inhibitors were reported in patients developing serious, including fatal, reactions, including rigidity, hyperthermia, myoclonus, altered mental status including excessive agitation, up to delirium and coma, unstable vital signs with possible rapid fluctuations. Also, these reactions were reported at the beginning of receiving monoamine oxidase inhibitors shortly after discontinuation of Non prescription Escitalopram with selective serotonin reuptake inhibitors.
In some cases, the symptoms were similar to neuroleptic malignant syndrome. Therefore it is impossible to combine escitalopram and monoamine oxidase inhibitors, or assign it within the first 14 days Non prescription Escitalopram their cancellation. Upon completion of a course of therapy with escitalopram and needed a break, at least 14 days prior to receiving monoamine oxidase inhibitors. The doctor, who appoints escitalopram for long-term treatment, should periodically evaluate the usefulness of escitalopram for each patient, in spite of the fact that the efficiency of use of escitalopram for Non prescription Escitalopram therapy in the treatment of depression has been demonstrated in a placebo.
A combination of symptoms such as tremor, agitation, hyperthermia, myoclonus may indicate the development of serotonin syndrome. Therefore, to reduce the dose or cancel escitalopram should be gradually for at least 1 — 2 weeks. The therapy escitalopram reported cases of hyponatremia. Cases of hyponatremia and syndrome of inappropriate secretion of antidiuretic hormone were noted in the treatment of racemic citalopram. Escitalopram should be repealed in the development of mania.
There have been Non prescriptions Escitalopram of seizures during clinical trials of escitalopram. Therefore, Non prescription Escitalopram should appoint escitalopram in patients with a history of seizures. Limited clinical experience in the use of escitalopram in the presence of patients with comorbidity. It should be used with caution escitalopram in patients with diseases that are accompanied by violation of hemodynamics, or metabolism.
In patients with unstable heart disease or myocardial Non prescription Escitalopram was not carried out systematic observations, because these patients were excluded from clinical studies premarketingovyh. In animal shows low capacity citalopram cause addiction research. Not conducted systematic observations in humans to evaluate the feasibility of escitalopram induce tolerance, Non Prescription Escitalopram, abuse, physical dependence. In controlled trials of escitalopram number of elderly patients was insufficient to adequately assess the safety generic Zofran efficacy of escitalopram, depending on age. We can not exclude the possibility of increased sensitivity in some patients to escitalopram. Some patients with panic disorder at the beginning of therapy with escitalopram may be exacerbated anxiety, usually disappearing within the next 2 weeks of treatment.
To reduce the likelihood of developing anxiety is necessary to use low initial doses. Escitalopram can increase glucose levels in the blood in Non prescription Escitalopram mellitus, which may require changes in dosage of hypoglycemic agents, Non Prescription Escitalopram. During treatment with escitalopram is recommended to refuse to perform potentially hazardous activities requiring increased attention and psychomotor speed reactions including vehicle control. Pregnancy and breastfeeding The use of escitalopram during pregnancy is possible if the supposed effect of treatment for the mother above the possible risk to the fetus. Strictly controlled and adequate studies safety of escitalopram were not carried out in pregnant women. At all Non prescriptions Escitalopram, the toxicity observed for the maternal organism clinical signs, reduction in food intake and body weight gain.
In newborns who were exposed to escitalopram at the end of III trimester of pregnancy, developed complications which required tube feeding, respiratory support, prolongation of hospitalization.